molecular‑pathology.com
Breast · prognostic

EndoPredict

Gene-expression signature in early ER-positive, HER2-negative breast cancer. It answers whether endocrine therapy alone suffices or whether chemotherapy belongs with it.

ER+ / HER2− pre- and postmenopausal node-negative and -positive
What the report answers
EPclin scale from 0 to 7 with the threshold at 3.3 Horizontal axis from 0 to 7. The threshold at 3.3 separates low from high risk of distant metastasis.

You receive two numbers. The 12-gene Molecular Score measures gene expression in the tumour: eight tumour-associated genes, three reference genes, one control gene for DNA contamination. The EPclin score adds tumour size and nodal status. It ranges from 0 to 7; at 3.3 runs the boundary between low and high risk.

The EPclin is what matters. In the ABCSG-6/8 cohorts — 1,702 patients, endocrine therapy only, followed for a median of 9.6 years — 62.6 percent fell into the low-risk group. Their risk of distant metastasis lay well below that of the high-risk group: hazard ratio 4.77 (95 % CI 3.37–6.67). In the premenopausal patients of a separate validation (n = 385) it was 3.58.

It becomes clinically interesting where score and marker diverge. In an analysis of 1,652 prospectively examined cases, EPclin and Ki-67 did correlate significantly. Even so, 118 of 449 patients with a Ki-67 above 20 percent were classified by EPclin as low risk — more than a quarter of those in whom the conventional marker would have advised chemotherapy.

This is not a contradiction but the whole point. The score measures something that grading and proliferation index do not capture. It does not replace them; it comes in addition.

What to send
Tissue
FFPE from the surgical specimen. The block or unstained sections, along with the H&E section.
Clinical details
Pathological tumour size and nodal status — both feed into the EPclin score. Plus ER and HER2 status.
What we take on
Assessment, annotation, enrichment. The tumour content is our problem, not yours.

Without tumour size and nodal status we cannot compute an EPclin, and the molecular score alone does not carry the decision. Please send both with the case — otherwise we have to ask, and the report is delayed by a day.

The report

We issue the report in English, signed by a specialist in pathology. It contains both scores, the threshold and the patient's placement.

One limitation we prefer to name ourselves: for the 21-gene and the 70-gene signature there are prospective studies (TAILORx, MINDACT). For EndoPredict the evidence is predominantly prospective-retrospective. The cohorts are large and followed for a long time, but it is a different level of evidence — something you should learn from us rather than have to find out yourself.

Turnaround
5 to 10 working days
from receipt of the sample
Accreditation
DAkkS · ISO 17020
References
  • Filipits M, et al. Prediction of Distant Recurrence Using EndoPredict Among Women with ER+, HER2- Node-Positive and Node-Negative Breast Cancer Treated with Endocrine Therapy Only. Clin Cancer Res. 2019;25(13):3865-72. PMID 31064782
  • Constantinidou A, et al. Clinical Validation of EndoPredict in Pre-Menopausal Women with ER-Positive, HER2-Negative Primary Breast Cancer. Clin Cancer Res. 2022;28(20):4435-43. PMID 36043530
  • Jank P, et al. Comparison of risk assessment in 1652 early ER positive, HER2 negative breast cancer in a real-world data set: classical pathological parameters vs. 12-gene molecular assay (EndoPredict). Breast Cancer Res Treat. 2022;191(2):327-33. PMID 34783927

A conversation before the first case

Material, fixation, and which clinical details we need. Half an hour now saves queries later.